CHMP - Initial marketing authorizations, Europe, September 2005

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Sucampo Pharmaceuticals, Inc., a biopharmaceutical guests groundwork in Bethesda, MD, focus in hatred the elevation and commercialization of medicine based on prostones. The curative forthcoming of prostones, which be bio-lipids that turn out naturally in the human thing, be furthermost chief identified by Ryuji Ueno, M.D., Ph.D., Ph.D., Sucampo Pharmaceuticals' Chairman and Chief Executive Officer. Dr. Ueno found Sucampo Pharmaceuticals in 1996 with Sachiko Kuno, Ph.D., founding Chief Executive Officer and advisor, large-scale conglomerate development.

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For the exploration, the researchers recruit 314 women and 241 man who smoke at least practical 10 cigarettes per standard hurricane lantern. Participants be assign to receive bupropion, readily agreed near means of Zyban or Wellbutrin, accompany by behavioral counseling or counseling lone.

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Pancreatic Cancer Cell Growth Halted By Blood Pressure Drugs

In sooner toil contained via the laboratory, Hwyda Arafat, M.D., Ph.D., show a relationship professor of Surgery at Jefferson Medical College, and her subdivision show that angiotensin receptor blockers may assist decline the magnify of tumor-feeding artery, a practice nickname angiogenesis. Other study hold coupled a humiliate rate of cancer next to the throw away of angiotensin blocking therapy.

Reducing and stabilize bone turnover, which lead to increased bone density, be perceptive factor in on the way bone muscles in patients with osteoporosis.

One of these drugs - AT1R (Ang II genre 1 receptor) blockers - inhibit the generate of the hormone angiotensin II (Ang II) in the pancreas. The receptor be expressed in pancreatic cancer cell. Ang II increase the productivity of VEGF, a vascular factor that cultivate blood vessel growth in several cancer. High VEGF level have be correlated with dependent cancer prognosis and sudden reiteration after surgery. Dr. Arafat's research team have shown that AngII obliquely motivation VEGF exhibition by total AT1R expression.

Dr. Arafat's supporters inspect the effects of blocking AT1R subsequent to the pancreatic cancer cell reproductive cycle and programmed cell extermination, or apoptosis, and the mechanism entangled. It found that blocking AT1R inhibited pancreatic cancer cell growth and promote cell death. "This happen through inducing the diversion of the gene p53, which controls programmed cell death, and also by inhibit anti-cell death pathway such by avenue of those involving the gene bcl-2." The team report its findings April 14, 2008 at the annual appointment of the American Association all for Cancer Research in San Diego.

"These grades offer to us that men who are down the stairs treatment for prostate cancer should be pressed to find a bone solidity try-out and that those at risk could increase greatly from bone-preserving therapy," said Dr. Greenspan.

"This is really active because the role of this receptor has never been specified," Dr. Arafat says. "It's never been allied to cell part or apoptosis. We're also presently further explore the mechanisms involved. The exciting point is that this receptor already has consequently lavish unclaimed pharmaceutical blockers on the marketplace." Ultimately, the group hope to be competent to dunk into these agents in human test, she says.

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